B. Nucleotide Excision Repair
C. DNA replication
D. Protein folding
This patient present feature of Xeroderma Pigmentosum
Ans. is ‘b’ i.e., Nucleotide Excision Repair
Exposure to UV rays damages DNA due to production of convalent linkages between adjacent pyrimidines. Normally the damaged DNA is repaired by excision and repair. In xeroderma pigmentosa the repair of UV damaged DNA is defective.