Author: Dr Abhishek Gupta

Its Better to Burn Out than to Fade !

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Anatomy
CLAVICLE
SHOULDER JOINT
HUMERUS BONE
RADIAL NERVE
MOVEMENTS OF SHOULDER JOINT
MEDIAN NERVE PALSY
ULNAR NERVE
MEDIAN NERVE
BRACHIAL ARTERY
AXILLARY ARTERY
MUSCLES OF PECTORAL REGION
TRIGEMINAL (V) NERVE
TRIGEMINAL NEURALGIA
MAXILLARY NERVE
MANDIBULAR NERVE
OPHTHALMIC (V1) NERVE
SCALP & SUPERFICIAL TEMPORAL REGIION
MUSCLES OF FACIAL EXPRESSION
BELL’S PALSY
MUSCLES OF MASTICATION
EXTRAOCULAR MUSCLES
FACIAL (VII) NERVE
Facial Nerve: Functional Components
GENICULATE GANGLION
AZYGOUS VENOUS SYSTEM
WALLS OF ORBIT
TEMPOROMANDIBULAR JOINT
SYNDROMES & DIAGNOSTIC TEST ASSOCIATED WITH FACIAL NERVE
MUSCLES OF TONGUE
FACE
LARYNX
LIGAMENTS OF LARYNX & EPIGLOTTIS
LARYNGEAL MEMBRANE & MUCOSA
MUSCLES OF LARYNX
MAXILLARY SINUS
FRONTAL SINUS
NASAL SEPTUM/ MEDIAL NASAL WALL
LATERAL NASAL WALL
FORAMENS OF SKULL
PALATE
Tympanic Membrane Of Ear/ EARDRUM
MIDDLE EAR
MASTOID ANTRUM
INTERNAL EAR
LARYNGEAL JOINTS & SPACES
Fluid Of Inner Ear
ANATOMY OF HEART
HEART-ARTERIAL SUPPLY, NERVE SUPPLY, VEINS OF HEART
LIVER
CHAMBERS OF HEART
SUPRAHYOID MUSCLES
INFRAHYOID MUSCLES
PERITONEAL FOLDS- LESSER OMENTUM & GREATER OMENTUM
Ventricular system
INFERIOR VENA CAVA
PERITONEAL CAVITY- LESSEER SAC & GREATER SAC
PORTAL VEIN
EXTRAHEPATIC BILIARY SYSTEM
INGUINAL CANAL
INGUINAL LYMPH NODE
KIDNEY- STRUCTURE, RENAL FASCIA & VASCULATURE
KIDNEY
Hypoglossal Nerve
OPTIC (II) NERVE
GLOSSOPHARYNGEAL NERVE
LUMBAR PLEXUS
SACRAL PLEXUS
SCIATIC NERVE
FEMORAL NERVE
Arterial Supply of Lower Limb
TIBIAL NERVE
VENOUS DRAINAGE OF LOWER LIMB
MUSCULAR COMPARTMENT OF LEG
HIP JOINT MOIVEMENTS
FEMORAL SHEATH
OBTURATOR NERVE
SAPHENOUS NERVE
FEMORAL TRIANGLE
HIP JOINT
ADDUCTOR CANAL
BONES OF FOOT
ANKLE JOINT- LIGAMENTS
MOVEMENTS OF FOOT & ANKLE
MUSCLES OF FOOT
LIGAMENTS OF KNEE JOINT
KNEE JOINT
SCIATIC FORAMEN
APPLIED ANATOMY OF MUSCLES OF UPPER LIMB
STOMACH
IMPORTANT FACTS ABOUT OSTEOLOGY OF HEAD & NECK
GENITOURINARY EMBRYOLOGY
ANTERIOR ABDOMINAL WALL
PHARYNGEAL APPARATUS
PHARYNGEAL/ BRACHIAL ARCH DERIVATIVES
DERIVATIVES OF ECTODERM
DERIVATIVES OF MESODERM
DERIVATIVES OF ENDODERM
GAMETOGENESIS- SPERMATOGENESIS
GAMETOGENESIS- OOGENESIS
THYROID GLAND
PARATHYROID GLANDS
PITUITARY GLAND
OLFACTORY (I) NERVE
VAGUS (X) NERVE
ACCESSORY (XI) NERVE
FACIAL NERVE INJURY
BONES OF HAND- CARPAL
BRACHIAL PLEXUS
ERB’S PARALYSIS
CARPAL TUNNEL SYNDROME
ANATOMY OF LUNGS
FISSURES & LOBES OF LUNGS
CONDUCTING SYSTEM OF HEART
AORTA
SUPERIOR VENA- CAVA
SUPRARENAL GLAND
URETER
FUNCTIONAL AREAS OF CEREBRAL CORTEX
CERVICAL VERTEBRAE
IMPORTANT JOINTS OF VERTEBRAL COLUMN
SPLEEN
PANCREAS
POSTERIOR TRIANGLE OF NECK
SUBMANDIBULAR GLAND
ANTERIOR TRIANGLE OF NECK
SEMICIRCULAR CANALS OF INNER EAR
CONTENTS OF TYMPANIC CAVITY/ MIDDLE EAR
MUSCLES OF PECTORAL REGION & AXILLA
NERVE SUPPLY OF MUSCLES OF UPPER LIMB
ULNAR ARTERY
RADIAL ARTERY
MUSCULOCUTANEOUS NERVE
AXILLARY NERVE
RADIAL NERVE PALSY
KLUMPKE’S PARALYSIS
BONES OF HAND- METACARPAL
BONES OF THE FOREARM- RADIUS
BONES OF THE FOREARM- ULNA
THORACIC DUCT
TRACHEA
OESOPHAGUS
EXTRAPULMONARY & INTRAPULMONARY BRONCHUS
ARTERIAL SUPPLY & VENOUS DRAINAGE OF LUNG
HILUM OF LUNG
COMMON PERIONEAL NERVE / FIBULAR NERVE
TIBIA BONE
FEMUR BONE
DIAPHRAGM
TONSILS (Palatine/ Faucial Tonsils)
WHITE MATTER OF CEREBRUM
EXOCRINE GLANDS

Physiology
SCAPULA
RENIN- ANGIOTENSIN SYSTEM
ANION GAP
ATRIAL NATRIURETIC PEPTIDE
SPERMATOGENESIS
PARATHROMONE
CHOLECYSTOKININ (CCK)
BASAL GANGLIA
PHYSIOLOGY OF EXERCISE
OXYGEN DISSOCIATION CURVE
SPIROMETRY
CEREBELLUM – DIVISIONS & FUNCTIONS
LIMBIC SYSTEM
HYPOTHALAMUS – DIVISIONS & FUNCTIONS
DISTURBANCES IN ACID-BASE BALANCES
ABSORPTION OF IRON
GLUCOSE TRANSPORTERS
PANCREATIC SECRETIONS
PRESSURE CURVES IN CVS
HEART SOUND
RENAL REGULATION OF ACID-BASE BALANCE
DIGESTION AND ABSORPTION OF PROTEIN
DIGESTION AND ABSORPTION OF FATS
DIGESTION AND ABSORPTION OF CARBOHYDRATE
STRETCH REFLEX
INSULIN – METABOLIC ACTIONS
ELECTRO-ENCEPHALOGRAM (EEG)
ANTI-DIURETIC HORMONE (ADH) / VASOPRESSIN
PULMONARY COMPLIANCE
NEUROTRANSMITTERS – FUNCTIONS & MECHANISM OF ACTION IN CNS
SECOND MESSENGER
RESTING MEMBRANE POTENTIAL
ACTION POTENTIAL
NERVE CONDUCTION
NERVE FIBER – CLASSIFICATION
SPINAL REFLEX & PROPERTIES
NEPHRON
INTERACTION BETWEEN CO2 & O2 TRANSPORT
PRODUCTION & REGULATION OF BILE
CEREBELLUM – NEURONAL CIRCUIT & DISORDERS
SURFACTANT
NEOCORTEX – ORGANIZATION & FUNCTIONS
HYPOTHALAMUS – NEURONAL FUNCTIONS
VENTILATION-PERFUSION RATIO (V/Q RATIO)
MEASUREMENT OF RENAL BLOOD FLOW
GLOMERULAR FILTRATION RATE (GFR) & FACTORS AFFECTING
REGULATION OF RENAL BLOOD FLOW
GLOMERULAR FILTRATION RATE (GFR) – MEASUREMENT
REGULATION OF GFR
GALL BLADDER – FUNCTIONS
MECHANISM OF RENAL TRANSPORT OF SODIUM, GLUCOSE & UREA
PERISTALSIS
RESPIRATORY RESPONSE TO EXERCISES
DIGESTION IN STOMACH – MOTILITY & GASTRIC EMPTYING
MINERALOCORTICOIDS – FUNCTIONS & REGULATION
GLUCOCORTICOIDS – FUNCTIONS & REGULATION
CALCITROPIC HORMONES
MENSTRUAL CYCLE
LACTATION
OVARIAN HORMONES
NEUROENDOCRINE RESPONSE TO STRESS
GASTROINTESTINAL HORMONES
SYNTHESIS OF THYROID HORMONE
GASTROINTESTINAL MOTILITY
DIGESTION IN SMALL INTESTINE – MOTILITY, SECRETION & CONTROL
DIGESTION IN LARGE INTESTINE – MOTILITY & REFLEXES
THYROID HORMONE – METABOLISM, TRANSPORT & REGULATION.
THYROID HORMONE – FUNCTIONS
SODIUM-POTASSIUM PUMP
CARDIAC MUSCLE CONTRACTIONS
SKELETAL MUSCLE CONTRACTIONS – CHARACTERISTICS
MUSCLE SPINDLES
SENSORY RECEPTORS
FEATURES OF SENSORY RECEPTORS
GENERAL EXTERORECEPTORS
THERMO-REGULATORY MECHANISM
INVERSE STRETCH REFLEX
SKELETAL MUSCLE CONTRACTION & RELAXATION -MECHANISM
SKELETAL MUSCLE PHYSIOLOGY – STRUCTURE & TYPES OF MUSCLE FIBERS
PROCESS OF SYNAPTIC INHIBITION
DEVELOPMENT OF POSTSYNAPTIC POTENTIAL
EXCITATION-CONTRACTION COUPLING
CYANOSIS
CARBON MONOXIDE POISONING
RESPIRATORY REFLEXES
CHEMICAL REGULATION OF RESPIRATION.
NEURAL MECHANISM OF BREATHING REGULATION
HYPOXIA
ALTITUDE PHYSIOLOGY
PHYSIOLOGY OF DEEP SEA DIVING
DEAD SPACE
RESPIRATORY PRESSURE
BLOOD PRESSURE
MEASUREMENT OF BLOOD PRESSURE
NEURAL REGULATION OF CVS
CARDIOPULMONARY REFLEXES
CARDIAC OUTPUT
PRINCIPLES OF BLOOD FLOW
ERYTHROCYTES
CARDIAC CONDUCTION
COAGULATION SYSTEM
HEMOSTASIS
CARDIAC CYCLE
PLASMA PROTEINS
HORMONES INFLUENCING RENAL ACTIVITY
GROWTH HORMONE
SPINAL CORD INJURY
PAIN AND ANALGESIA
G-PROTEIN COUPLED RECEPTORS
STAGES OF SLEEP
UPPER MOTOR NEURON Vs. LOWER MOTOR NEURON PARALYSIS
REGULATION OF FEEDING BEHAVIOR
BLOOD BRAIN BARRIER
CEREBROSPINAL FLUID
SPEECH
GUSTATION
RESPIRATORY FAILURE
OLFACTION
TRANSPORT MAXIMUM
MEMORY
LEARNING
THALAMUS
SALIVA
HORMONAL CHANGES AT PUBERTY
SEMEN
REABSORPTION & SECRETION OF PROXIMAL TUBULE
RENAL TRANSPORT OF POTASSIUM, CALCIUM & UREA
RE-ABSORPTION & SECRETION IN DISTAL TUBULE
GASTRIC ACID SECRETION – MECHANISM & PHASES
REABSORPTION & SECRETION IN LOOP OF HENLE
GASTRIC ACID SECRETION – REGULATION & OUTPUT
CHEMICAL REGULATION OF CVS
REABSORPTION & SECRETION IN COLLECTING DUCTS
COMPOSITION OF ALVEOLAR AIR
PHYSIOLOGY OF PITUITARY GLAND
INSULIN – RECEPTORS AND REGULATION OF SECRETION
MOTOR CORTEX
REGULATION OF THIRST
REGULATION OF POSTURE
ASSOCIATION AREAS
CELLULAR ELEMENTS OF CNS
RETICULAR FORMATION
LUNG VOLUMES & CAPACITY
SYMPATHETIC Vs. PARASYMPATHETIC NERVOUS SYSTEM
MOLECULAR MOTORS & MARKERS
CYTOSKELETON
COMPONENTS OF BODY FLUIDS
OSMOSIS
SMOOTH MUSCLE CONTRACTION – IMPORTANT FACTORS & FEATURES
INTERCELLULAR JUNCTIONS

Biochemistry
HMP Shunt
Glutathione
ETC
Gluconeogenesis
Urea Cycle
Purine Salvage Pathway
Vitamin A
Vitamin A Deficiency
FISH(Fluorescent in situ hybridization)
Glucokinase
Tay-Sachs disease
Maple Syrup Urine Disease
Cholesterol Biosynthesis
Malate Shuttle
Vitamin C
Collagen
Trace element:Iodine
Trace element : Iron
Trace element:Zinc
Trace element : Copper
Trace element : Fluorine
Trace element :Selenium
NIACIN
Proline
Histidine
Carnitine
Hartnup Ds
Niemann–Pick disease
Bile Pigment
Krebs Cycle
Ketone Bodies
Classification of Amino Acid
Antioxidant
Chemistry of Carbohydrates
glycolysis cycle
Tryptophan
Phenylketonuria
Nucleotides
Gout- Purine metabolism disorder
LESCH-NYHAN SYNDROME
Severe combined immunodeficiency
STRUCTURE OF DNA
Different Types of DNA
Denaturation of DNA
Orotic aciduria
Regulation of gene expression
Antimetabolites
Albinism
properties of amino acids
Codons and genetic codes
INHIBITORS OF PROTEIN SYNTHESIS
COENZYMES
CLASSIFICATION OF ENZYMES
REGULATION OF ENZYME
ISOENZYMES
ENZYME MARKERS
Lipids
Fatty acids
ESSENTIAL FATTY ACIDS
Isomerism in fatty acids
TRIACYLGLYCEROL
Phopholipids
Sphingophospholipids
Gaucher’s disease
BIOMEMBRANES
De Novo Fatty Acid Synthesis
Regulation of Fatty Acid Synthesis
Oxidation of Fatty acids
Regulation of fatty acid synthesis
Different types of fatty acid oxidation
Metabolism of VLDL
Fatty liver
Hyperlipoproteinemia
Bile Acid Synthesis
Lipoprotein- structure
Hypolipoproteinemia
Regulation of cholesterol synthesis
Vitamin A
Vitamin A deficiency
Vitamin D
Vitamin D- Deficiency
Vitamin E & deficiency
Vitamin K & Deficiency
Vitamin B1 & Deficiency
Vitamin B2 & Deficiency
Vitamin B3
Vitamin B3 & Deficiency
Vitamin B6 & Deficiency
Vitamin B7 (Biotin)
Vitamin B9 (Folic Acid)
Vitamin B12 & Deficiency
Bile Acid Synthesis
Lipoprotein- structure
Vitamin C Deficiency- Scurvy
Classification of Minerals
Structure of Heme
Biosynthesis of Heme
Metabolism of Bilirubin
Structure of Hemoglobin
Denaturation of Protein
Separation techniques of protein
Disease associated with collagen
Protein Folding
Ubiquitin
Prion Disease
Plasma Cell Disorder
Ehler’s- Danlos Syndrome
Alport’s Syndrome
Polysaccharides
Mucopolysaccharides (Glycosaminoglycan)
Mucopolysaccharidosis
Rapoport Leubering cycle

Microbiology
Streptococcus
Streptococcus Pneumonia
Streptococcus Pyogens: Morphology,Transmission, Antigenicity and Lysogeny
Streptococcus Viridans
Human Immunodeficiency Virus (HIV)
Replication & Transmission Of HIV
Diagnostic Techniques In HIV
AIDS Control Programmes
WHO clinical staging of HIV/AIDS for children with confirmed HIV infection
Prevention Methods In HIV
Treatment Modalities In HIV/AIDS Patients
HIV Involving Nervous System
Toxoplasma gondii: Toxoplasmosis
LUNGS IN HIV
AIDS defining Opportunistic infections and Neoplasms
Kidney In HIV
Eye, Endocrine system, Hematopoietic system, CVS and Dermatological system In HIV
Oral and GIT Manifestations Of HIV
Chlamydia
Diagnosis & treatment Of Chlamydia Infection
Vibrio cholerae
Vibrio cholerae Culture & Diagnosis
Vibrio Cholera & It’s Clinical Features
Vibrio Cholerae Virulency
Vibrio Cholera Transmission & Cholera Control
Vibrio Cholerae Management
Structure Of Immunoglobulin
Immunoglobulin A
Immunoglobulin M
Immunoglobulin E
Immunoglobulin G
E.coli:Structure and Virulency
STAPHYLOCOCCI
Staphylococcus aureus: Pathogenesis, Clinical Syndromes
Staphylococcus aureus: Diagnosis, Prevention and Treatment
Corynebacterium Diphtheria
Mycobactreium Tuberculosis
Salmonella Typhi
Clostridium Perfringens
Clostridium Tetani
Clostridium Botulinum
Clostridium difficile
Herpes Simplex Virus
Prion
Polymerised Chain Reaction
Epstein Barr Virus
Hepatitis B Virus
Shigella
Mycobacterium leprae
Leprosy- Epidemolgy,Vaccination,NTLEP
Rabies Virus
Rabies :Transmission and Clinical features
Rabies :Diagnosis and Negri bodies
Rabies: Pre-exposure and Post-Exposre Prophylaxis
Lepra Reaction And Erythma Nodosum Leprosum
Leprosy- Diagnosis
Leprosy:WHO Classification ,WHO Disability grading
Leprosy Classification-Madrid and Ridley and Jopling Classification
Streptococcus Pyogens: Clinical manifestation, Diagnosis and Treatment
Streptococcus Pneumonia :Clinical Manifestations , Diagnosis and treatment
Chlamydia trachomatis
Chlamydia pneumoniae and Chlamydia psittaca
SYNTHESIS OF IMMUNOGLOBULINS
HUMAN IMMUNOGLOBULINS CLASSES
E. coli: Clinical manifestations
E. coli Diagnosis and Treatment
Staphylococcus aureus: Morphology,Culture and Biochemical reaction
Staphylococcus aureus: Virulence and Toxin
COAGULASE – ve STAPHYLOCOCCI
Corynebacterium Diphtheria: Clinical manifestation, Complications, Diagnosis and Treatment
Shigella : Clinical Findings, Pathogenesis, Lab Diagnosis and Treatment
Clostridium Perfringens: Clinical manifestation, Diagnosis and Treatment
ANTIGEN ANTIBODY REACTION AND PRECIPITATION REACTION
AGGLUTINATION
TYPE I HYPERSENSITIVITY
TYPE II HYPERSENSITIVITY
TYPE III HYPERSENSITIVITY
TYPE IV HYPERSENSITIVITY
INTERLEUKINS
Neisseria gonorrhoeae :Basics
Neisseria gonorrhoeae : Clinical manifestations
Neisseria gonorrhoeae :Diagnosis and Treatment
Clostridium tetani: Clinical manifestations
Clostridium tetani : Diagnosis, treatment and Prevention
Bacillus Anthrax
Bacillus Anthrax:Clinical manifestation, Diagnosis and treatment
Bacteriophage
Proteus bacilli
Bordetella pertussis
Bordetella pertussis: Clinical manifestations and diagnosis
Brucella
Brucellosis
Rickettsiae
Treponema Pallidum
Treponema Pallidum :Syphilis
Treponema Pallidum: Diagnosis and Treatment
Parvovirus
Pox Virus
Influenza virus
Influenza virus: Clinical manifestations, Complications, Diagnosis, Treatment and Prevention
Avian Influenza
Respiratory Syncytial Virus (RSV)
Rotavirus: Classification, Structure, composition and properties
Rotavirus: Pathogenesis, Clinical symptoms, laboratory diagnosis, and Treatment
Hepatitis A Virus
Hepatitis C Virus
Hepatitis E and Hepatitis D
Dengue Fever
Chikungunya
Candida
Candida: Clinical Manifestations
Candida: Diagnosis and Treatment
Pneumocystis jiroveci Pneumonia
Histoplasma capsulatum
Entamoeba histolytica
Entamoeba Histolytica: Clinical Manifestations
Entamoeba Histolytica: Diagnosis and Treatment
Leishmania
Dry Heat Sterilization
Moist Heat Sterilization
Mucormycosis
Adenovirus
Aspergillosis
Nocardia
DERMATOPHYTES
Blastomycosis
Mumps Virus
Measles
Measles- Vaccination and WHO’s measles elimination strategy
Giardia lamblia
Giardiasis
Cryptococcus neoformans
Cryptococcosis
Neisseria Meningitidis
Campylobacter jejuni
Bacillus cereus
Toxoplasma gondii
Pulse Polio and Polio eradication
Polio Virus
Poliomyelitis
Plasmodium
Plasmodium: Clinical Features (Malaria)
Plasmodium: Diagnosis
Malaria: Treatment
Haemophilus influenzae
Bordetella pertussis: Control of whooping cough
Nipah virus
Sterilization Methods( Radiation,Chemical Agents and Sporicidal Agents)
Disinfectant
Hemolytic Streptococci- Group A, B, C, D and F
Actinomyces and Actinomycosis
H. aegyptius and H. ducreyi
Helicobacter pylori
Non-Venereal Treponematosis- Bejel, Yaws and Pinta
Transfer of genetic information
Acanthamoeba
Naegleria fowleri
Trypanosoma cruzi and Trypanosoma brucei
Leptospira
Mycoplasma
Mycobacteria other than Tuberculosis-MOTTS/Atypical mycobacteria
Legionella
Pseudomonas aeruginosa
Vibrio – V. parahaemolyticus, V. alginolyticus, Vibrio Vulnificus, Vibrio Mimicus
Pasteurella multocida
Borrelia burgdorferi and Lyme disease
Borrelia and Relapsing Fever
Sporothrix schenckii
Coccidioides immitis
Cestodes(Tapeworms)
Varicella Zoster
Cytomegalovirus (CMV)
Coxsackievirus
Ebola Virus
Differences between Exotoxins and Endotoxins
K. pneumoniae, K. ozaenae and K.rhinoscleromatis
Yersinia pestis
Human PapillomaVirus (HPV)
Culture medium
Genetic Mechanism of Drug Resistance
Innate Immunity
Adaptive/Acquired Immunity
Lymphoreticular system

Pharmacology
Haloperidol
GLYCERYL TRINITRATE
Digoxin
Atropine
Octreotide
BROMOCRIPTINE
Halothane
Thiopentone
Propofol
Isoflurane
Sevoflurane
Fluoxetine
Clozapine
Lithium
Heparin
Pilocarpine
Sodium Cromoglycate
Leukotriene Antagonist
Acyclovir
Nitric Oxide
Linezolid
zidovudine
Cisplatin
Drugs causing gynaecomastia
Trastuzumab (Herceptin)
PYRAZINAMIDE
Rifampicin
Ethambutol
Aminoglycoside
Anti tubercular treatment regimen
Metronidazole
Ketoconazole
CHLORAMPHENICOL
AZITHROMYCIN
Sulphonamide
COTRIMOXAZOLE
DOXYCYCLIN
TETRACYCLIN
Dapsone
PENICILLIN
CLASSIFICATION OF ANTIMICROBIALS
Amoxicillin
Cephalosporin
AChE inhibitors
OP Poisoning
Glaucoma Pharmacological Management
Epinephrine
Beta adrenergic receptor antagonists or beta-blockers
Insulin
DOPAMINE
ORDER OF KINETICS
First pass metabolism
Cholinergic receptor
Alpha adrenergic receptor
Enzyme inhibition (competitive, non-competitive and uncompetitive)
Glucocorticoids
Mineralocorticoids
Aspirin
K+sparing diuretic
Loop Diuretics
Paracetamol
PHENYTOIN
Benzodiazipines
Anti-Parkinsonism Drugs
Histamine & Histaminic receptors
Monoclonal Antibody
Valproic acid (Sodium Valproate)
Anti-thyroid Drugs
Cyclosporin
TICLOPIDINE
INFLIXIMAB
DAPAGLIFLOZIN
SYMPATHETIC RECEPTORS
DOSE RESPONSE CURVE
THERAPEUTIC DRUG MONITORING
ANTI-CHOLINERGIC DRUGS
DRUG DISTRIBUTION
NEWER ANTI-DIABETIC DRUGS
ORAL HYPOGLYCEMIC DRUGS

Forensic Medicine
Criminal Procedure Codes
Lead poisoning
Postmortem Caloricity
IPC
INQUEST
CONSENT
The Transplantation of Human Organs Act – 1994
PROFESSIONAL NEGLIGENCE (MALPRAXIS)
FUNCTIONS OF INDIAN AND STATE MEDICAL COUNCILS
Consumer Protection Act, (CPA/COPRA) – 1986
EUTHANASIA
EXHUMATION
CORPUS DELICTI
Drowning-Autopsy findings
Drowning
Drowning- Test
Types of firearm
Bullet and Gunpowder
Fingerprint
Firearm: Identification of Weapon
Firearm wounds
Firearm:Tests, Phenomenons and Suicide VS Homicide
Laceration
Summon
Late Postmortem changes – Adipocere
Late Postmortem changes – Putrefaction
Late Postmortem changes – Mummification
Organophosphates Poisoning
Mercury poisoning
Sex Determination-Nuclear Sexing
Sex Determination by Skeleton
Age Determination- Teeth
Determination of Age
AGE IDENTIFICATION FROM BONES
Age determination of Fetus
Embalming
Incised wounds
Abrasion
Contusion/Bruise-Types
Contusion/Bruise-Factors and Color changes
Medical Evidence
Witnesses and Recording Of Evidence
Rigor mortis
Postmortem changes -Immediate
Postmortem changes – Early
Motor Vehicle injuries
Explosion injury
Stab wound
Regional Injuries
Hanging- Types, Causes of Death, Postmortem Findings and Medicolegal Importance
Pedestrian Injury
Injuries: Medicolegal Aspects
Physical Torture
Inquest
PROFESSIONAL MISCONDUCT (INFAMOUS CONDUCT)
Medical negligence
Professional secrecy and Privileged communication
Race Determination
Superimposition and Tattoo Mark
Somatic ,molecular and Sudden Death.

Pathology
Pulmonary Tuberculosis
Genital Tuberculosis
Genitourinary Tuberculosis
Gastrointestinal Tuberculosis
Skeletal Tuberculosis
Tuberculous Meningitis
Tuberculosis Of Skin
Diagnostic Techniques In Tuberculosis
Typhoid
TP53
Apoptosis
Sickle Cell Anemia
Langerhan’c cell Histiocytosis
Transforming growth factor Beta
ANCA(Antineutrophil Cytoplasmic Antibody)
Granulocyte Colony Stimulating Factor(G-CSF)
Hemophilia
Aplastic Anemia
Thalassemia
Paraneoplastic Syndrome
Sebaceous cyst
Pulmonary Embolism
Primary hyperaldosteronism
Wilson’s disease
Systemic Lupus Erythematous
CYSTIC FIBROSIS
Cellular adaptation
Reversible cell injury
Irreversible cell injury
Necrosis
Necroptosis
Autophagy
Intracellular accumulations
Pathological clacification
Cellular ageing
ACUTE INFLAMMATION- Vascular events

ENT
CSF Rhinorrhoea
Malignant Otitis Externa (ENT)
Pinna / Auricle
Otosclerosis / Otospongiosis
Septoplasty
Atrophic rhinitis / Ozaena
Vasomotor Rhinitis
Acoustic Neuroma / Vestibular Schwannoma / Neurilemmoma / 8th Nerve tumor
Rhinoscleroma
Choanal atresia
Frenzel Glasses
Cauliflower Ear
Menier’s Disease
Pinna
External Auditory Canal
Auditory Ossicles
Tympanoplasty
Malignant Otitis Externa
BERA
Acoustic reflex
Complications of Otits Media
Lateral Sinus Thrombophlebitis
Atrophic Rhinitis
Allergic Rhinitis
Acute Sinusitis
Complications of Sinusitis
Fungal Sinusits
Acute Tonsilitis
Tonsillectomy
Quinsy
Adenoid
Ludwig’s Angina
Epistaxis
Tracheostomy
Laryngo-tracheo-bronchial foreign bodies
CSF Rhinorrhea
Nasopharyngeal Angiofibroma
Snoring and Obstructive Sleep Apnea
Acute Epigottitis
Acute Layngo-tracheo-bronchitis
Fracture of Hyoid Bone
Fracture of Temporal bone
Fracture of the facial bones-Nose,Maxilla,Mandible,Zygomatic
Glomus Jugulare
Layngomalacia
Carcinoma of Tongue
Carcinoma of Larynx
Carcinoma of Oral Cavity
Maxillary Carcinoma
Nasopharyngeal Carcinoma
Retropharyngeal and Parapharyngeal Abscess
Laryngeal Paralysis
Laryngeal Nerve Supply
Tubercular Laryngitis
Neonatal Airway
Laryngeal involvement in miscellaneous causes.
Subglottic Stenosis
Recurrent Laryngeal Papillomatosis
Benign Lesions of Larynx
Voice Disorders
Ethmoidal Polyp
Antrochoanal Polyp
Thyroplasty
Laryngocele
Rhinosleroma
Rhinosporidiosis
FESS
Physiology of hearing (ENT)
Special Hearing Tests
Auditory Pathway

Ophthalmology
Retinitis Pigmentosa
KERATOCONUS
Blow out Fracture
Sympathetic ophthalmitis
OPHTHALMIA NEONATORUM
CRVO & CRAO
REFSUM DISEASE
GLAUCOMA
EALES DISEASE
THYROID OPTHALMOPATHY
VITREOUS HEMORRHAGE
DIABETIC RETINOPATHY
CONTACT LENSES
APHAKIA
ASTIGMATISM
MYOPIA
ANATOMY OF CORNEA
HISTOLOGY OF CORNEA
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Gurukul Tradition – Lesson of Humility & Hardship

Important Disclaimer –
Anything I write is to share some interesting and worth sharing insights.
I have no intention to hurt / harass / defame / oppress any person/ sex / caste / religion.
If I do , forgive me as it was completely unintentional. 


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Gurukul

(Lesson of Humility & Hardship)

I often wonder why Kids Prefer Cartoon, Grandparents (and Parents or equivalent) prefer Likes of Ramayan and We Students stuck at SPLITSVILLA.

But this is not what I will write about this week.

This Saturday my Parents were watching Ramayan – The Old Ramanand Sagar one

[I really wonder if any one of you reading this will know about Old Ramayan from Ramanand Sagar but For Simplicity, The One created by Ramanand Sagar besides being very very old , is high on content and low on cinematography as compared to many recent ones]

The Episode was in which Lord Ram , had now attained 5 years of age and was not to got to “Gurukul” to learn along with his other 3 brothers.

Now To Understand this situation fully  consider these following

  1. Lord Ram (and his brothers) were born after very tough penance by Lord Dhasratha
  2. Lord Ram was the heir to the BIGGEST and the STRONGEST Kingdom of all times
  3. Lord Ram was Only 5 Years (or so) old when he had to goto gurukul.

Now Lets See what Gurukul meant at that time.

  1. Beg for food (For Yourself and your Guru)
  2. Do all the work in Gurukul (Remember @ 5 Years of Age)
  3. Live on minimal

 

So You see this is what I found interesting.

Lord Ram , who was born out of great penance , and was the Heir to the Strongest Kingdom on earth was sent to gurukul by his Father , wherein he will have to beg for Food, Live with other students like ordinary with no special privileges , work his A**E off , all at a tender age of 5.

 

 Now How many of us will do this to our Kids ??

 

In fact , we go out of our way to provide the BEST of BEST for our KIDs.

We teach them to make “Good” Friends a.k.a Other Children who belong to the same social strata.

We Give them access to everything which we can afford [even if we have to take loans ]

 

So can we Learn a Few things from Those “Gurukul” days or is it just that now “Things are Different” ??

Well I feel, Still we can Learn a Few Things

 

Lesson of Humility

 

In ancient scriptures it is said that to learn from the Master , the Disciple must sit at the teachers feet. This may be just another way of saying that , HE Who is not HUMBLE will not learn anything. The lesson of Humility was etched into the fabric of Gurukul. [Begging for Food, Staying like ordinary ]

Now,  I know neither we have Gurukuls nor we can send our children to Beg . But Still we should try to incorporate the value of Humility into our children.

 

Importance of Hardships

 

I know we all want to give everything to our children. We want to pave every path and carve every opportunity for them. We never want them to face any hardships in Life.

But

These hardships are the forces that will sculpture them into masterpieces.

These hardships are the forces that will make them strong enough to face any and every challenge in Life

These hardships will build their character and their will to find a way out of every situation.

These hardships will bring out the innermost strength and talent out of them.

 

Imagine a Cub being brought up in a Cage with all facilities

but no challenges [of the wild],

Will he be able to survive in a Jungle ??

Even Dogs may hunt him down.

 

if You have read till this Point , I have a challenge For you.

Can you think of and share with me some practical ways to inculcate HUMILITY in our Children ??

Email me at abhishek@medicoapps.org

 


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Previous Week Email –

The Paradox of Choices – Why More is not better ? Click Here to Read

 

Be Awesome
Stay Awesome
Dr Abhishek Gupta

 

The Paradox of Choice – Why More is not Better ??

Important Disclaimer –
Anything I write is to share some interesting and worth sharing insights.
I have no intention to hurt / harass / defame / oppress any person/ sex / caste / religion.
If I do , forgive me as it was completely unintentional. 


Weekly Newsletter Banner Ad 2- Above Heading


The Paradox of Choice

Why More is not Better ??

(The JAM Experiment)

 

We Love Choices .

Choices are Good .

More Options to Choose From , Happier We are .

And How Can We Forget Amazon Ads – Aur Dikhao —- Aur Dikhao


 

 

But What if I Tell You

” More Options is Not Good ”  .

Will You Believe me.

I tell you a personal story.

I was in Class 8E (Computer Section). And in our section we had 2 Girls [I Guess Computer was not that HOT Then]

What really surprised me was that both the Girls , were far more in sought after than girls in other sections.  This was really surprising for me then and a mystery to me till last week when I read about the JAM Experiment.

 

In 2000, psychologists Sheena Iyengar and Mark Lepper from Columbia and Stanford University published a study about jams.

On a regular day at a local food market, people would find a display table with 24 different kinds of jams. Then on another day, at that same food market, people were given only 6 different types of jam choices.

 

Now Guess

On Which Day There was a More Interest on the JAM Table ?? (Means More people went to the JAM Table0

On Which Day there was more sales of the JAM bottles ?? (Means more people bought the JAM 0

 

Well Here is the Result.

On the Day there were 24 Different JAM bottles showcased, significantly more people went to see the JAM tables.

On the Day there were only 6 Different JAM bottles , Significantly more people bought the JAM Bottle.

 

Why Does this happen??

 

This Happens because of what Psychologists call as “CHOICE Paralysis”

 

The above experiment was then repeated in different circumstances and with a lot many variations BUT still the results were same.

More Choice – More Interest

More Choices – Less Action

 

But Here is where THING GET Interesting.

 

What was more clear in all the experiments that when the number of choices were more , even when people who bought the JAM bottles , they were LESS SATISFIED and LESS HAPPY with their Purchase.

 

Whaaaaaaaaaaaaaaaat………………..

Yes people were more satisfied and happy with there choices when they had to choose between 6 JAM Bottles than they were when they had to choose between 24 JAM Bottles [Mind You the 6 Bottles Types were also there in 24 Bottle set]

 

Now this is What I call Interesting.

What this means that if the number of choices are more people will talk more about it , Take less action , and even when they take action they will be less happy and satisfied

 

How does this Effect you in REAL Life  ??

You Know what is the Most Common Question asked to me by students.

“Sir Which is the Best Book for XYZ Subject ??” or “From Which Book should we Study”

In my time [Way back in 2006], this was never the question.

There were Only 2 Books , Mudit Khanna & Amit Asish [Several Volumes] and Anyone who could complete it is sure to get into AIIMS.

But Now For Each Subject I guess there are more than 3-5 Choices .

Results – Even 1-2 Years into preparation , students will still ask this question from me.

So What Should You Do ??

Now you cannot stop Authors from publishing books. But If you realize this , You can decide on any one book and just focus all your time and attention on it [Even though you may keep feeling that it is not the best resource.]

 

Other Interesting Places where you can Observe the same thing.

It is easier to “Hook Up ” or find your life partner in a causal party than on Tinder or Bharat Matrimony.

(More Choices , More Interest but Less Action0

 

One advice which I regularly give students [who find it difficult to  revision] is to keep 15-20 pages which he plans to revise next morning by his bed and start revising as soon as he gets up the next morning.

Zero % Choice 

100 % Action 

500 % Happiness / Satisfaction.

 

Now To really use this remember , wherever you feel unhappy / unsatisfied , you reduce [or completely remove] the number of choice and then go with the Only choice.

Initially you may feel that you have not taken the “BEST” Decision but if you stick with your choice for some time , soon it will become the “BEST” Decision for you and you will be really happy and satisfied with you choice.

 

So Now Hope You understand the MYSTERY why those 2 Girls for SECTION 8E got all the Attention [and were asked out so often]

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Be Awesome
Stay Awesome
Dr Abhishek Gupta

 

 

Previous Week Email –

Dopamine – Can You Use this Addiction hormone to Your AdvantageClick Here to Read

 

 

 

 

 

Dopamine – Can You Use This Addiction Hormone to Your Advantage

DOPAMINE

Can You Use this Addiction Hormone

to Your Advantage ??

 

Hey There Freedom Fighter,

Last Week I when I wrote about How to Tackle Personal & Family Problem , and among 1000’s of reply I got there was an interesting reply from Dr Annu.

While the email itself had so many practical Tips on tackling personal & family problem , there was a really interesting mention of

“Give Yourself the Pleasure of “Target Done” Shots.

.

 

This Single Idea If you can understand will Help you to really get more things done and having great fun doing it.

So Here I am writing about it in this Tuesday’s Personal Letter to you.

 

Five Hormones that Control Us (Literally)

  1. Endorphins [Endurance Hormone]
  2. Dopamine [Reward or Pleasure Hormone]
  3. Serotonin [Leadership Hormone]
  4. Oxytocin [Trust / Safety Hormone]
  5. Corticosols [Stress Hormone]

 

I know you may be knowing the physiological role of these hormones but TRUST me there is little that you know about the psychological effects of these hormone.

Not Only you will enjoy learning the psychological aspects of these hormones but also  change the WAY you LOOK at these hormone.

I will discuss about these hormones one by one in my upcoming Tuesday’s Letters to Freedom Fighters.

 

Understanding Dopamine

from Evolutionary Point of View

 

First a FUN Fact – Dopamine Hormone is Exclusive to Human Beings and many consider that it may be JUST the Factor that separates Humans from other species on this planet.

So Whats So Different about this HORMONE ??

Essentially DOPAMINE forms the Main Neurotransmitter for the REWARD System of the Brain.

The main psychological function of this Hormone is to give high psychological incentive to do activities which are routine but extremely essential for survival.

Activities like Eating , Having Sex , completing some work.

Now In Hormonal Hierarchy When Dopamine Exists in the Body, the psychological effects of no other hormone matters.

When Everything is in Balance , this is a Beautiful Hormone.  Dopamine gives you the extra incentive to go and complete routine works with more enthusiasm.

But When there is an Dopamine Dependence [As in cases of Alcoholism , Drug Addiction , Smokers, Gambling ] , the dopamine will completely hijack your system and make you do things that are even harmful.

 

Such is the Power of Dopamine.

Power of DOPAMINE 

Have You Ever wondered why are you so addicted to your SMART Phones ??

Well Guess What, All the smart phone makers and top app makers [Facebook , Whats app, Twitter, Instagram, etc ] have designed their products and services to HACK Your Brain Dopamine System and get you addicted.

You get a Email Notification – Ding . You Open the Email . Ding Dopamine Release . You Feel Good

Somebody Liked your Photo . You get a Notification . DING  . Dopamine Release . You Feel Good

Somebody Sends you a Whats App Forwarded Message . Notification . Ding . Dopamine Release . You Feel Good

So much is the behavior ingrained in you that if you do not get any notification , you open your facebook app and check for any new updates . You Open Your Facebook App . DING . Dopamine Release . You Feel Good.

 

Imagine you are driving your car. You get a Whats App Notification . You will reach your destination in another 10 Minutes. And You know that the Whats App Message cannot be anything important (and most probably some stupid forwarded joke or fake news) .

But Just at the next red light or for many while still driving you open your app to see whats the new message.

Ding. Dopamine Release . You Feel Good.

 

You are all motivated to Study Hard for NEET PG Preparation . You now start looking for all books , apps, programs. And You Find Medicoapps 1800 Pages Notes. Wow , this 1800 Pages can Help You complete you syllabubs in next 6 Months.

You Hit on the Buy Now Button . Complete the Purchase . DING . Dopamine Release . You Feel Good 

(And Even You know that you have all the necessary review books, dams notes and other stuff packed under you study table and so these notes will be a utter waste for you)

 

Controlling the Power of DOPAMINE 

By Now You Must have realized the Power of Dopamine.

This is a WILD Beast which if you learn to Tame will be the single biggest reason for your success in almost anything.

Before trying to control and modulate the Dopamine Release Lets see the Hidden Factors behind it. Just as a caution the below mentioned details are about self meditated release of dopamine and may or may not be applicable to release /level of dopamine due to drugs / nicotine / alcohol, etc.

  1. Dopamine Release is Stronger to Visual Clues- Creation of Visual Clues
  2. Dopamine Release is Pleasure Hormone – Making Boring things pleasurable

Lets Understand Both these one by one

 

Dopamine & Visual Clues 

Self Meditated release of Dopamine is secreted more when you are clearly able to see the results. Remember the Notification pop up on your mobile when someone likes your photo on Facebook or when someone sends  you a Whats App message.

Visual clues can be really make or break when it comes to DOPAMINE.

That is the reason people put pictures of Ferrari , Ali Bhat , Ranbir Kappor on Wall (Whenever you see it, Ding – Dopamine Release . You Feel Happy).

So Advice for You .

Put A Visual Clue for Your Goals

Want to Get Radiology Seat in PG ??? Get a Photograph of a Radiologist Working and Paste it where you study.

Want to Be One of the Top Ranker in PG . Take Photograph of toppers of Previous Year and Make it your Screensaver on Mobile

Yes the Most Important Thing. Daily Routines.

Plan your day and Write it in a piece of paper. Research has clearly proven that if you want to achieve any goal , write it down.

You may already have in your mind what all you plan for tomorrow. But Dont deny your HARD WORKING and SUPPORTING Brain of much needed Dopamine.

Write down your Daily Plans [And You can keep doing to even after you clear you PG Exams]

 

Now Lets Discuss the Second Aspect

 

Dopamine is a Pleasure Hormone

Now another most important thing to remember is that Dopamine is a Pleasure Hormone.

So No matter what you do , If you cannot make it pleasurable , Good Bye Dopamine.

So I must be Joking. How can anyone make STUDYING Pleasurable.

Well Here are a Few Tips

  1. Break it into smaller pieces. Remember Every Small Piece Complete is DING. Dopamine Release . You Feel Happy. So if your target is Read 20 Pages of Biochemistry Tomorrow. All You will get is ONE Shot of Dopamine Release. But Instead of Making your target your as 20 pages of Biochemistry to Read 1> Amino Acid Classification – 2> Amino Acid Properties  3> Amino Acid Absorption 4> So on and So forth , so that the you have just listed all the topics in the same 20 pages , Look What You have Done. With Each Small Topic you complete and Tick off the List , You get a DING. Dopamine Release . You Feel Happy.
  2. Make it Easy to Do. Always Do easy things first . Because Once the Dopamine is released it will keep you doing more and more to get more Ding . Dopamine Release . You Feel Happy Cycle. So Start your day with something really simple and then move to harder tasks.
  3. Give Yourself Some Real Rewards Once you complete it. Almost No body like the taste of Alcohol, but everyone drinks it for the BUZZ which comes after drinking it. So Even though you may not thoroughly enjoy studying but associating it with something you enjoy at end of every task completed will make the entire study enjoyable. One such way is by Ticking the DONE Items. Everyone Enjoys Ticking (Right !!!) Also Like I love Coffee . So After Every Morning Exercise I make myself a HOT Home Brewed Coffee. And if I do not exercise any day , I do not drink coffee that day. There are days I exercise for 5 Minutes only to get myself my Coffee. You can tie your Study Schedule to Facebook. So Every Item you complete on Your Todo list , You get 10 Minutes on Facebook / Instagram. 
  4. Any More More Innovative Ways you can think of

 

There are so so many students / people I know that are already using the above hack to trick their brains into doing more.

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Hope You liked and will benefit from this article.

 

I would Love to Know Your Thoughts and also if I should write such analysis on Other 4 Psychological Hormones .

Email me at abhishek@medicoapps.org

 

Be Awesome
Stay Awesome
Dr Abhishek Gupta

 

 

Previous Week Email –

How to Tackle Family & Personal Problems during NEET PG Preparation – Click Here to Read

How to Tackle Family and Personal Issues ???

How to Tackle

Family & Personal Issues

during NEET PG Preparation ??

 

I keep calling Students to understand to understand on deepest level what are the major challenges they are facing during their NEET PG Preparation.

I expected students to talk about their difficulty in completing the huge syllabus or difficulty in revision and remembering facts  or other such problems.

But Astonishing as it may sound , many students talk about how their are completely surrounded by some personal or family issues and hence the NEET PG Preparation itself has taken back seat.

At First I use to ignore this and move on, but over last few years I have realized how almost every students who is preparing for NEET PG is at some level facing so many difficult situations at personal or family level.

While Some are able to tackle these issues many students are completely unaware how to move forward.

 

Lets Face it ,

Life was much simpler when we were Kids.

 

Our Biggest worry was getting pass marks(for most of us) Half-yearly and Final exams.

Our Favorite Food was “Maggie”

Our Favorite Actor was “Shah Rukh Khan” and Favorite Actress was “Kajol”

Our Biggest Disappointment was when we lost the ball (while playing cricket) and the game had to be stopped till we collected enough money to buy another ball.

And Our Only RESPONSIBILITY was ……. ??????

Responsibility ?? What the Hell was that ??

But Then We Got into medical college and without realizing we are almost 23 Years Old

(By the Way Do you realize that you have spent your entire Sweet TEENage preparing for Medical College and then Attending medical college )

 

What Happens at 23 Years

(And More So if You are a doctor )

 

Life will never be simple again.

You eat maggie almost like a meal many times a week.

Favorite food or favorite anything does not really mean anything to you.

And Yes suddenly there is so many people (Family , Family of family , Friends , Friends of Friends , Relatives , Relatives of Relatives , People Whom You know , People Whom you do not know) who suddenly have a lot of expectation from you.

You are expected to take decisions.

And So many things that it is almost impossible to even list 10% of things here.

Soon it feels like this


 

 

So is it really surprising that NEET PG takes a Backseat.

 

So what can we do about it ??

 

Now the First Step is realizing that LIFE will get even more complex as we grow older.

We cannot do many things about it.

But Here is a small Tip (From my personal experience)

I always wake up at 5:00 in Morning. Yes Even On Sundays. This is a habit I acquired long back. And I would start my day with doing 2 Hrs of doing the most important things for the day.

So when I was preparing, I would study everyday for 2 Hrs.

Now When I am running Medicoapps, Every day from 5:00 to 7:00 AM I will create Videos / Modules / Reply to Emails.

5:00 -7:00 AM Time for me is 

GOD Hours.

 

Now Lets See Why this 5:00 – 7:00 AM is Absolutely different from the Rest of the Day.

  1. You are absolutely fresh after having a Good Night Sleep
  2.  You are most relaxed [Most of the Fire Fighting and Anxiety comes as we move along the day]
  3. NOBODY Disturbs you during these Hours
  4. This Time BELONGS Exclusively to you

 

Throughout the Entire Day , Anybody can HIJACK your time with their own work or agenda but Nobody has an access to these morning 5:00-7:00 AM .

This BELONGS EXCLUSIVELY to You

 

So What Can we Do in this Time ??

  • You can use it in any thing which is important to you.
  • You can Use it to start your NEET PG Preparation.
  • You can use it to exercise [If Health is a priority for you]
  • You can use to Learn Guitar or write poems.

 

How Much can you achieve using these 2 hrs??

I would say pretty much anything.

I am a full time practising physician and all what I have done in Medicoapps is 90% Done during these GOD Hours.

Research, Says that we always over-estimate what we can achieve it in short term and under-achieve what we can achieve in Long Term.

Just Two Hrs a day , Can help you build the required momentum to Get the ball moving.

 

And What can be a better way to start your day other than doing something that you truly want to accomplish.

 

But This is not All.

 

The REAL MAGIC happens what happens throughout the day.

With such a Kick Start and taking care of the Most Important thing even before the day starts for most of your friends,

You have are filled with a COMPLETE different level of energy to tackle your entire day with whatever the challenges it may bring.

Just Imagine That.

Now by giving such a simple solution (but a very powerful one ) I am in no way trying to underestimate the challenges you are facing at personal and family level because I understand things can get really really complex.

But Still thing simple solution may help focus on your most important goals along with effectively tackling your day to day challenges.

 

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I would Love to Know Your Thoughts . Email me at abhishek@medicoapps.org

 

 

Be Awesome
Stay Awesome
Dr Abhishek Gupta

 

Image Based Question-68079

Question

Stenosis ofArtery (Box) is NOT seen in

A. Atherosclerosis.

B. PAN.

C. Takayasu arteritis.

D. Fibromusucular dysplasia.

 

Show Answer

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